Dietary tyrosine suppresses the rise in plasma corticosterone following acute stress in rats
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Cited by (34)
Metabolomics reveals distinct neurochemical profiles associated with stress resilience
2017, Neurobiology of StressCitation Excerpt :Rats given tyrosine before acute tail-shock displayed neither shock induced norepinephrine depletion nor the deficits in exploratory behaviors observed in saline-treated animals (Reinstein et al., 1984). Similarly, pre-treatment with tyrosine not only prevented behavioral depression and norepinephrine depletion after acute restraint stress and intermittent tail-shock but also suppressed the rise in plasma corticosterone (Reinstein et al., 1985). Additionally, we found that dominant hamsters had lower serine concentrations in the BLA/CeA compared to control hamsters.
Elevation of rat brain tyrosine levels by phenelzine is mediated by its active metabolite β-phenylethylidenehydrazine
2014, Progress in Neuro-Psychopharmacology and Biological PsychiatryCitation Excerpt :The rationale for treatment of depression with tyrosine includes increased production of NA and enhanced resilience against the HPA-mediated stress response. In rats exposed to an acute stressor, pre-treatment with tyrosine prevented NA depletion and behavioral deficits (Reinstein et al., 1984), as well as suppressed the rise in plasma corticosterone levels (Reinstein et al., 1985). Studies on human volunteers demonstrated that tyrosine supplementation alleviated decrements in working memory and psychomotor tasks, adverse moods and performance impairment following the environmental stress of cold exposure (Banderet and Lieberman, 1989; Mahoney et al., 2007; O'Brien et al., 2007; Shurtleff et al., 1994).
Dietary l-tyrosine alleviates the behavioral alterations induced by social isolation stress in mice
2009, Brain Research BulletinCitation Excerpt :There was no significant main effect of rearing condition or interaction between rearing condition and diet. Some reports indicate that l-tyrosine ameliorates the changes incognition and behavior caused by acute stress by preventing a reduction of NE in response to acute stress [2,19,29]. However, the effectiveness of l-tyrosine on stress was limited.
Altered amino acid homeostasis in subjects affected by fibromyalgia
2009, Clinical BiochemistryTyrosine improves behavioral and neurochemical deficits caused by cold exposure
2001, Physiology and Behavior