A double mutation in the androgen receptor allows prostate cancer cells to become responsive to glucocorticoids. This discovery sheds light on a new pathway by which prostate cancer cells can escape androgen dependence (pages 703–706).
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Brinkmann, A., Trapman, J. Prostate cancer schemes for androgen escape. Nat Med 6, 628–629 (2000). https://doi.org/10.1038/76194
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DOI: https://doi.org/10.1038/76194
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