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Viral, worm and radical implications for apoptosis

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References (17)

  • M. Scheffner et al.

    Cell

    (1993)
  • D.P. Lane

    Current Biol.

    (1992)
  • G.R. Pearson

    Virology

    (1987)
  • D.M. Hockenbery

    Cell

    (1993)
  • M. Miura

    Cell

    (1993)
  • J. Yuan

    Cell

    (1993)
  • C.A. Ray

    Cell

    (1992)
  • H.M. Ellis et al.

    Cell

    (1986)
There are more references available in the full text version of this article.

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