Aspirin-induced asthma: Hypersensitivity to fenoprofen and ibuprofen in relation to their inhibitory action on prostaglandin generation by different microsomal enzymic preparations
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Group 2 innate lymphoid cells are recruited to the nasal mucosa in patients with aspirin-exacerbated respiratory disease
2017, Journal of Allergy and Clinical ImmunologyCitation Excerpt :Previous work has shown that AERD can be mediated by innate type 2 immunity. First, the potency of the COX-1 inhibition determines the severity of the reaction, which indicates that it is not adaptive recognition of a specific drug structure that activates the immune system.30 Second, 2 danger signals released from epithelial cells, IL-33, an alarmin-like cytokine, and TSLP, an IL-7–like cytokine, have been shown to be upregulated in nasal polyps from patients with AERD and important drivers for COX-1 inhibitor–induced reactions.7,8,31
Respiratory symptoms in patients with inflammatory bowel disease and the impact of dietary salicylates
2007, Digestive and Liver DiseaseCitation Excerpt :Additionally, therapeutic agents of value in one condition can prove useful in the other (e.g. corticosteroids, cromoglycate) [17]. Many asthmatics are allergic to salicylates, with acute reductions in peak expiratory flow rate (PEFR) and overt bronchoconstriction, usually after exposure to aspirin [18]. Aspirin-induced asthma is increasingly shown to be important in the adult population [19].
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2002, Revista Portuguesa de PneumologiaA clinical study of the impact on consumer health of dyes used in Tunisia
1990, Revue francaise d'allergologie et d'immunologie cliniqueInhaled lysine-aspirin as a bronchoprovocation procedure in aspirin-sensitive asthma: Its repeatability, absence of a late-phase reaction, and the role of histamine
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