Retrograde transport of horseradish peroxidase in transected axons. 3. Entry into injured axons and subsequent localization in perikaryon
References (29)
What is the signal for chromatolysis?
Brain Research
(1970)- et al.
A study of the dynamics of retrograde transport and accumulation of horseradish peroxidase in injured neurons
Brain Research
(1975) - et al.
Retrograde transport of horseradish peroxidase in transected axons. 1. Time relationship between transport and induction of chromatolysis
Brain Research
(1974) - et al.
Retrograde transport of protein tracer in the rabbit hypoglossal nerve during regeneration
Brain Research
(1972) Outflow from cut ends of nerve fibres
Exp. Cell Res.
(1956)On axoplasmic flow
Int. Rev. Neurobiol.
(1975)The horseradish peroxidase technique applied to the teleostean nervous system
Brain Research
(1975)- et al.
Afferents to the rat caudoputamen studied with horseradish peroxidase. An evaluation of a retrograde neuroanatomical research method
Brain Research
(1974) - et al.
Subcellular accompaniments of axon reaction in cervical motoneurons of the cat
The distribution within the brain of ferritin injected into the cerebrospinal fluid compartments. I. Ependymal distribution
J. Cell Biol.
(1965)
Die Verteilung exogener Peroxydase Im Endoneuralraum
Histochemie
(1971)
The smooth endoplasmic reticulum: structure and role in the renewal of axonal membrane and synaptic vesicles by fast axonal transport
Brain Research
(1975)
The early stages of absorption of injected horseradish peroxidase in the proximal tubules of mouse kidney. Ultrastructural correlates by a new technique
J. Histochem. Cytochem.
(1966)
Evidence for recycling of synaptic vesicle membrane during transmitter release at the frog neuromuscular junction
J. Cell Biol.
(1973)
Cited by (127)
The impact of short-lasting repeated vibrations on retrograde axonal transport, the expression of CGRP and parvalbumin in lower lumbar dorsal root ganglia
2011, Brain ResearchCitation Excerpt :How do vibration-stimulated primary sensory neurons increase a retrograde axonal transport? One of the possible explanations is that repeated vibrations can up-regulate neuronal retrograde transport capacity via increased non-specific influx into axotomized neurons and subsequent transport to the perikaryon (Kristensson and Olsson, 1976). It was well documented that vascular spasticity is a direct result of vibration (Curry et al., 2002; Welsh, 1980).
Neuronal injury increases retrograde axonal transport of the neurotrophins to spinal sensory neurons and motor neurons via multiple receptor mechanisms
1998, Molecular and Cellular NeurosciencesThe ratio of pre- to postganglionic neurons and related issues in the autonomic nervous system
1995, Brain Research ReviewsEfficacy of seven retrograde tracers, compared in multiple-labelling studies of feline motoneurones
1994, Journal of Neuroscience Methods
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