Rose bengal activates the Ca2+ release channel from skeletal muscle sarcoplasmic reticulum☆
References (32)
- et al.
J. Mol. Cell. Cardiol
(1984) - et al.
J. Mol. Cell. Cardiol
(1984) - et al.
Arch. Biochem. Biophys
(1988) J. Biol. Chem
(1970)- et al.
J. Biol. Chem
(1984) - et al.
J. Biol. Chem
(1987) - et al.
J. Biol. Chem
(1989) - et al.
- et al.
Biophys. J
(1986) J. Biol. Chem
(1984)
J. Biol. Chem
J. Biol. Chem
J. Biol. Chem
Arch. Biochem. Biophys
Biophys. J
Am. J. Physiol
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This work was supported by grants from the American Heart Association (87-915), the American Cancer Society (CH-445), and NIH (1R15GM44337-01) to J.J.A.; by American Heart Association Grant (87-1065), and the Western Pennsylvania Heart Association to G.S.; and by NIH (ES05002) to I.N.P. This is Environmental Sciences and Resources Publication 264.
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Present address: Department of Pharmacology, Oregon Health Sciences University, Portland, OR 97201.